Preliminary reports suggest conolidine may inhibit unique ion channels, reducing neuronal excitability and limiting discomfort signals. This mechanism is particularly relevant in neuropathic soreness, wherever abnormal signaling leads to persistent discomfort. Furthermore, conolidine seems to affect G protein-coupled receptor (GPCR) pathways integral to discomfort notion. Computationa... https://anthonya062arh9.shoutmyblog.com/profile